By Ramana S. Moorthy MD
Starts with an in-depth review of immunemedicated eye illness, summarizing easy immunologic techniques, ocular immune responses and specific subject matters in ocular immunology. Discusses the scientific method of uveitis and stories noninfectious (autoimmune) and infectious varieties of uveitis, with an extended part on viral uveitis and new fabric on infectious and noninfectious scleritis. improved detection of infectious brokers by means of immunologic and genetic equipment and new biologic therapeutics are special. additionally covers endophthalmitis, masquerade syndromes, problems of uveitis and ocular features of AIDS. features a variety of new colour photos. significant revision 2011-2012
Read or Download 2011-2012 Basic and Clinical Science Course, Section 9: Intraocular Inflammation and Uveitis (Basic & Clinical Science Course) PDF
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Additional resources for 2011-2012 Basic and Clinical Science Course, Section 9: Intraocular Inflammation and Uveitis (Basic & Clinical Science Course)
This shift requires a molecular change of the immunoglobulin heavy chain class, and this is regulated by specific cytokines released by the helper T lymphocyte. For example, treatment of an antigen-primed B lymphocyte with IFN -y induces a switch from IgM to IgGI production. Treatment with IL-4 induces a switch from IgM to IgE production. Role of regulatory T lymphocytes The immunoregulatory role of regulatory (or suppressor) T lymphocytes has become partially clarified, especially through the induction of immunomodulatory cytokine synthesis by regulatory T lymphocytes.
And -C) serve as the antigen-presenting platform for CD8 T lymphocytes (Fig 2-2). CD8 T lymphocytes include natural killer T cells and regulatory T cells. Class I molecules are present on almost all nucleated cells. In general. class I APCs are best for processing peptide antigens that have been synthesized by the host cell itself. including most tumor peptides or viral peptides after host cell invasion. MHC class II molecules (ie. HLA-DR. -DP. and -DQ) serve as the antigen-presenting platform for CD4.
The Th2 subset of DH cells secretes IL-4, IL-S, and other cytokines. IL-4 can induce B lymphocytes to synthesize IgE, and IL-S can recruit and activate eosinophils within a site. IL-4 can also induce macrophage granulomas in response to parasite-derived antigens. 2-3 Ocular Inflammatory Diseases Thought to Require a Major Contribution of Th1-Mediated DH Effec10r Mechanisms Site Disease Presumed Antigen Conjunctiva Contact hypersensitivity to contact lens solutions Giant papillary co njun ctivitis Phlycten ulosis Chronic allog raft rejection Marginal infiltrates of blepharitis Disciform keratitis after vira l infection Acute anterior uveitis Sarcoidosis-associated uveitis Intermediate uveitis Sympathetic op hthalmia Vogt-Koyanagi- Harada syndrome Birdshot retinochoroidopathy Acute thyroid orbitopathy Giant cell arteritis Thim erosa l or other chemicals Cornea and sclera Anterior uvea Retina and choroid Orbit Unknown Bacteria l anti gens Histocompatibility antigens Bacterial antigens Viral antigens Uveal autoantigens.